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Microglial Phagolysosome Dysfunction and Altered Neural Communication Amplify Phenotypic Severity in Prader-Willi Syndrome with Larger Deletion

Felipe Correa-da-Silva,Jenny Carter,Xin-Yuan Wang,Rui Sun,Ekta Pathak,José Manuel Monroy Kuhn,Sonja C. Schriever,Clarissa M. Maya-Monteiro,Han Jiao,Martin J. Kalsbeek,Pedro M. M. Moraes-Vieira, Johan J. P. Gille,Margje Sinnema,Constance T. R. M. Stumpel,Leopold M. G. Curfs,Dirk Jan Stenvers,Paul T. Pfluger,Dominik Lutter,Alberto M. Pereira,Andries Kalsbeek,Eric Fliers,Dick F. Swaab,Lawrence Wilkinson,Yuanqing Gao,Chun-Xia Yi

Acta Neuropathologica（2024）

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Key words
Hypothalamus,Fornix,Microglia,Immunosurveillance,Myelin,Glymphatic system,Oxytocin
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