Dormancy is an innate survival mechanism seen across taxa. The intracellular pathogen and a major public health concern, Plasmodium falciparum, utilizes dormancy to evade drug pressure and recrudesce to cause recurrent infections that facilitate the spread of resistance genes. Mechanisms underlying selection pressure of genes conferring Artemisinin resistance and the interaction of the innate survival mechanism of dormancy on the acquisition of inheritable resistance remains elusive. My disserta