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Carole Torsney is a Senior Lecturer in the Centre for Discovery Brain Sciences at the University of Edinburgh. Carole Torsney received her PhD from University College London as part of the Wellcome Trust 4 year PhD programme in Neuroscience with Professor Maria Fitzgerald. She then undertook postdoctoral training in the Department of Physiology and Cellular Biophysics at Columbia University, New York with Professor Amy MacDermott. She was then awarded a Caledonian Research Foundation Fellowship, then a Senior Academic Fellowship followed by Lectureship at the University of Edinburgh. She is interested in studying the abnormal functioning or pathophysiology of the sensory nervous system in pain syndromes, primarily using electrophysiological approaches and is a member of the Edinburgh Translational Pain Research Group.
My research group investigates the neural mechanisms and circuitry that mediates chronic pain. Specifically we are interested in defining the afferent-spinal circuits that mediate the symptoms of touch evoked pain (allodynia) and exaggerated pain (hyperalgesia) with the aim of identifying novel therapeutic targets. We are also interested in exploring these questions in both sexes given there is increasing recognition of sex differences in pain sensitivity and chronic pain susceptibility but poor understanding of the underlying basis. Interestingly we have recently demonstrated that the temporal relay of pain signals, by C fibres to the spinal cord, is altered in a sex and injury dependent manner.
As part of the University of Edinburgh Translational Pain Research Programme I also collaborate with clinical colleagues from the Edinburgh Cancer Research Centre to study chemotherapy-induced neuropathic pain. Notably, this neuropathic pain is so poorly managed that some cancer patients stop life prolonging chemotherapy. We have explored interventions to reduce the oxidative stress and mitochondrial damage that is thought to underlie these conditions. This work has established that antioxidants that are specifically targeted to mitochondria are able to limit the development of chemotherapy-induced neuropathic pain. This includes melatonin, which has a low toxicity profile and is well tolerated in humans and so has strong potential for translation to patients
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论文共 39 篇作者统计合作学者相似作者
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The journal of pain/Journal of pain (2024)
Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature (2019)
Faculty Opinions – Post-Publication Peer Review of the Biomedical Literature (2019)
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#Papers: 39
#Citation: 1540
H-Index: 19
G-Index: 30
Sociability: 5
Diversity: 3
Activity: 5
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