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Using molecular imaging methods to measure neuroinflammation in vivo, his group has shown persisting microglia activity after stroke is a risk factor for accelerated neurodegeneration and poor motor recovery (Radlinska 2009 and 2010, Thiel 2010). Combining these molecular PET imaging approaches with diffusion MRI methods, they were the first to demonstrate in stroke patients in vivo that these degenerative changes are not limited to the affected tracts but cause microstructural changes in previously unaffected fibres (Radlinska 2013). His group was the first to image in vivo the morphological correlate of transcallosal disinhibition (Paquette 2011, Thiel 2006), a major mechanism of potential maladaptive neuroplasticity during the recovery phase after stroke (Anglade 2014). Further important progress has been made in establishing image analysis methods for high-resolution 18F-FMZ PET imaging to directly assess cortical neuronal density in vivo (LaFougere 2011 , Funck 2014, 2018), demonstrating a relationship between cortical thickness and the density of neuronal structures in the cortex of normal elderly subjects depends on regional cytoarchitechonics (LaFougere 2011) and can be used to image selective neuronal loss in the penumbra of stroke patients (Funck 2014,2017) and other neurological diseases (Pomares 2017,2019).
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Neurorehabilitation and neural repairpp.15459683241245410-15459683241245410, (2024)
The Canadian journal of neurological sciences. Le journal canadien des sciences neurologiquespp.1-36, (2024)
Alzheimer's & dementia : the journal of the Alzheimer's Association (2024)
EJNMMI radiopharmacy and chemistryno. 1 (2023): 33-9
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Franziska E Hildesheim, Alexander N Silver, Adan-Ulises Dominguez-Vargas,Justin W Andrushko,Jodi D Edwards,Numa Dancause,Alexander Thiel
Frontiers in rehabilitation sciences (2022): 795335
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