Location of Pathogenic Variants in PSEN1 Impacts Progression of Cognitive, Clinical, and Neurodegenerative Measures in Autosomal‐dominant Alzheimer's Disease Stephanie Schultz , Zahra P. Shirzadi , Aaron Schultz , Lei D. Liu , Colleen Fitzpatrick , Eric R. McDade , Nicolas Barthelemy , Alan Renton , Bianca Esposito , Nelly Joseph-Mathurin , Carlos D. Cruchaga , Charles Chen , Alison Goate , Ricardo Francisco Allegri , Tammie L. S. Benzinger , Sarah C. Berman , Helena M. Chui , Anne R. Fagan , Martin C. Farlow , Nick A. Fox , Brian S. Gordon , Gregory R. Day , Neill J. Graff-Radford , Jason J. Hassenstab , Bernard Hanseeuw , Anna R. Hofmann , Clifford R. Jack Jr , Mathias M. Jucker , Celeste A. Karch , Robert Koeppe , Jae-Hong I. Lee , Allan Levey , Johannes N. Levin , Ralph Martins , Hiroshi C. Mori , John Morris , James J. Noble , Richard Perrin , Pedro P. Rosa-Neto , Stephen Salloway , Raquel R. Sanchez-Valle , Peter Schofield , Chengjie A. Xiong , Keith J. Johnson , Randall A. Bateman , Reisa P. Sperling , Jasmeer Chhatwal Aging Cell（2023）
Key words
Autosomal dominant Alzheimer disease (ADAD), heterogeneity, neurodegeneration, Presenilin-1, PSEN1
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