The ECM and CAFs interact to differentially regulate PDAC parenchymal (CPC) and CSC phenotypes to determine pancreatic ductal adenocarcinoma (PDAC) progression

Stefania Cannone, Maria Rafaella Greco,Hélène Guizouarn,Olivier Soriani,Richard Tomasini,Valeria Casavola,Katrine Zeeberg,Stephan Joel Reshkin,Rosa Angela Cardone

semanticscholar（2019）

引用 0|浏览1

暂无评分

摘要

Abstract Summary CAFs and acellular stromal ECM components interact to drive metastatic progression by stimulating the hallmark behaviors of each tumor cell type that contribute to metastasis: invasion in the CPCs and growth and angiogenesis in the CSCs.

查看译文

AI 理解论文

溯源树

样例

生成溯源树，研究论文发展脉络

Chat Paper

正在生成论文摘要