Cardiac myosin-binding protein c phosphorylation enhances cardiac relaxation
Journal of the American College of Cardiology(2013)
摘要
Cardiac myosin-binding protein-C (MyBPC3), a thick filament protein that inhibits myosin-actin interaction to slow cross-bridge cycling, has been implicated in diastolic dysfunction. MyBPC3 phosphorylation releases this inhibition; thus, we hypothesize that MyBPC3 phosphorylation promotes lusitropy
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