Asprosin aggravates vascular endothelial dysfunction via disturbing mitochondrial dynamics in obesity models.
Obesity (Silver Spring, Md.)(2023)
摘要
The findings demonstrate that ASP causes and even exacerbates vascular endothelial dysfunction through promoting mitochondrial fission in obesity, suggesting that ASP can act as an early predictive marker of blood vessel dysfunction and become a novel potential therapeutic target for obesity-related cardiovascular diseases.
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关键词
endothelial dysfunction,vascular endothelial dysfunction,mitochondrial dynamics,asprosin,obesity
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